Smegma is a normal, harmless secretion — shed skin cells, the skin’s own oils and moisture — that collects under the foreskin in men and around the clitoral hood in women. It is not an infection, not an STI, and not evidence of poor hygiene.
I say that first because of who reads a page like this. In my Chennai clinic the men asking me about smegma are almost never ill. They are teenagers who found something white under the foreskin, married men convinced they caught something, fathers holding a nine-month-old with a yellow lump. Almost none were ever shown how to wash their own genitals — men’s genital hygiene is rarely discussed despite mattering for men’s health (Mizelle, 2026). A gap in what men get told, not a personal failing.
So what most people need first is not treatment. It is permission to stop panicking. If you would rather just have it looked at, that is a two-minute job.
The short version
In a nutshell
It is a secretion, not dirt
Desquamated epithelial cells — skin the inner foreskin and glans shed continuously (Baral, 2023).
It is not an STI and not contagious
You did not catch it and you cannot give it to anyone.
Everyone with a foreskin makes it, daily
The prepuce protects the glans from abrasion and trauma throughout life and supplies the skin needed for erection (Dobanovacki, 2012).
The smell is bacterial, not the smegma
The subpreputial space is normally colonised by a wide range of organisms (Chung, 2019).
Daily washing is the whole treatment
Normal washing with soap and water plus gentle retraction is all an uncircumcised penis needs (McGregor, 2007). Always roll the foreskin forward afterwards.
It has never been shown to cause cancer — but hygiene is not irrelevant
The one study everybody quotes could not tell whether the smegma caused the disease or followed it, and said so itself (Brinton, 1991). Poor genital hygiene over years is a listed modifiable risk factor (Uppal, 2026) — an argument for washing, not for fear.
Read those six if nothing else. The rest answers what patients ask me next.
Quick facts
- Smegma is opaque white or pale yellow and is largely shed epithelial cells (Baral, 2023).
- It adds no extra microbiological risk — in boys, where it has been directly compared, the subpreputial space is colonised whether or not smegma is there (Demir, 2020).
- Over 75% of boys referred to specialists for foreskin problems have a normal foreskin (Sutton, 2023).
- Women produce smegma too, in the labia minora and under the clitoral hood.
- High blood glucose promotes yeast growth and blunts local immunity, and candidal balanitis occurs almost exclusively in uncircumcised men (Nyirjesy, 2013).
What smegma actually is
Composition. Desquamated epithelial cells — skin the inner foreskin and glans shed continuously — is what the published description names (Baral, 2023). It also carries the skin’s own oils and moisture, which is ordinary dermatology rather than anything this small literature measured. No pus, no semen, no urine. When a man asks me what it is made of, I give him one line: shed skin and skin oil, nothing more.
Appearance. Thick, white or pale yellow, soft and crumbly, like paneer. I use paneer because every patient I say it to knows exactly what I mean. In infants it can collect into a yellowish-white lump — a smegma pearl — which is benign, and the treatment is reassurance (Baral, 2023).
Function. It is not waste. What is documented is the job of the structure it sits under: the prepuce protects the infant’s glans from faeces and ammonia in nappies, protects the glans from abrasion and trauma throughout life, and provides sufficient skin in erection (Dobanovacki, 2012). That a moist mucosal surface behaves better than a dry one is standard physiology; no study here measures smegma itself as a lubricant, so I will not pretend one does.
The word is Greek for soap — whoever named it was describing texture, not filth.
Where smegma forms
In men: in the subpreputial space between the inner foreskin and the head, especially in the coronal groove. It needs that fold, so it affects uncircumcised and partially circumcised men.
In women: around the clitoral hood and between the inner and outer labia. Female smegma is normal and almost never discussed, so women mistake it for discharge or thrush — and the vulva has many normal anatomic variations and benign lesions that mimic disease (Thakker, 2026). I include this section because wives and partners ask me about it in the same consultation, usually right at the end.
In circumcised men: far less, but not zero. Skin still sheds and makes oil, so the exposed head still needs washing. Circumcision shifts the local bacterial population (Onywera, 2020); it does not abolish the material.
Why smegma smells
Let me explain what’s actually happening, because almost everyone gets this backwards. Smegma on its own is close to odourless. The smell comes from bacteria.
That space is warm, humid and enclosed — in uncircumcised men the moist, warm space under the foreskin is described as one that promotes yeast growth, especially when hygiene is poor (Nyirjesy, 2013). It is normally colonised by bacteria too: in one prospective study of asymptomatic uncircumcised boys, 31 bacterial species were isolated from smegma (Chung, 2019), and similar work in Kano isolated Staphylococcus epidermidis, Staphylococcus aureus and Escherichia coli from the same site (Anyanwu, 2012). Colonisation is the normal state of that space with or without smegma — exactly what the comparative study found (Demir, 2020).
Bacteria breaking down accumulated material is what produces the odour: general microbiology, not a finding from these swab studies, none of which measured smell. My patients call it cheesy, musky, or like sour milk. I tell every one of them the same thing: it settles within a few days of daily washing, it needs no medicine, and it is not evidence that anyone did anything wrong.
Left too long, accumulation does more than smell. Balanoposthitis is one of the common preputial complications the foreskin-care literature exists to prevent (Leeson, 2025), and cleaning the space daily is what prevents it.
Why you might be producing so much smegma
Infrequent or incomplete washing is the commonest reason — it is made daily, so if it is not removed daily it collects into a paste. Anatomy matters too: deeper folds trap more.
A tight foreskin. If it will not retract fully you cannot clean the space. That is phimosis, and in an adult it deserves assessment rather than harder scrubbing.
Heat and sweat. The Chennai variable, and no international page weights it properly. In a hot, humid climate, in tight or layered clothing, you produce more and it smells sooner. Patients who move abroad tell me the problem “went away”. The climate changed; they did not.
Hormones and age. Sebaceous activity rises with androgens at puberty — ordinary physiology, not a finding from the smegma literature — which is why most boys first notice it in adolescence.
Poorly controlled diabetes. High blood glucose promotes yeast attachment and growth and interferes with immune responses, and candidal balanitis occurs almost exclusively in uncircumcised men (Nyirjesy, 2013). When a man over 35 has recurrent buildup and a foreskin tightening over months, I check his sugars first.
Most of what follows is reassurance. The two places where it is not — the cancer section and the red-flag table — are marked plainly, and you should read those properly.
Is this smegma or an STI?
Smegma is not a sexually transmitted infection, not an infection of any kind, and not contagious. There is nothing foreign there to transmit. In my clinic, I see this every week: a man who is certain he has caught something, who has already spent a week frightened, and who needs one look and one sentence. A few real problems do look similar at a glance, so here is the discriminator I use.
| What it is | How it looks | Wipes away? | Symptoms | What to do |
|---|---|---|---|---|
| Smegma | Soft white paste in the groove or labial folds | Yes | None | Wash daily |
| Candidal balanitis | Red sore glans, white curd-like patches | Redness stays | Itch, soreness | Antifungal treatment (Stary, 1996) |
| Pearly penile papules | Ring of tiny bumps around the rim | No — anatomy | None | Nothing |
| Fordyce spots | Pale grains inside shaft or scrotal skin | No — anatomy | None | Nothing |
| STI discharge | Fluid from the urethral opening, not the groove | Returns in hours | Burning, sores | Get tested |
| Lichen sclerosus | White, thickened, scarred skin at the tip | No | Tightening, splitting | Specialist review (Barry, 2026) |
Pearly penile papules and Fordyce spots are normal structures rather than findings — that is why those two rows carry no reference and nothing to do. Fluid coming from the urethra is a different problem entirely, and worth reading about separately, whether it is discolouration you have noticed in semen or the discharge of an infection like chlamydia.
The lichen sclerosus row is the one that matters. Male genital lichen sclerosus is repeatedly reported in the literature as candidal or other infective balanitis, and attributing it to a positive swab without specialist input delays the diagnosis that counts (Barry, 2026). White, stiff skin is neither smegma nor thrush.
The single most useful test is the one in that middle column, and it is the one I perform first in the room: smegma wipes away and stays away until it rebuilds. Almost nothing else on the list does.
Does smegma cause cancer? The honest answer
Here’s the honest answer. Smegma is a normal physiological secretion and has never been shown to cause penile cancer. Neither the current French AFU guideline nor the most recent review of the disease lists it among the risk factors (Neuville, 2024; Uppal, 2026). I have written the rest of this section the way I would say it across a desk, because the correct conclusion is not “nothing matters” — it is “wash properly, and know what a lesion looks like”. Over-reassuring you here would be its own kind of failure.
Where the claim came from. A case-control study of 141 men with penile cancer and 150 community controls in a high-risk area of China. Conditions restricting the movement of the foreskin — phimosis and paraphimosis — were strongly related to risk. Poor hygiene practices also appeared to increase risk, “particularly as evidenced by detection of smegma on physical examination”. In the same sentence the authors flagged the limit of their own finding: it was “difficult to decipher whether this association was etiologic or merely a consequence of disease” (Brinton, 1991). They closed by calling for further evaluation of the role of hygiene in the causation of penile cancer.
So: a real positive association, direction unresolved by the people who found it, and that is still where it sits. Direction matters, because a man with an obstructing, non-retractile, cancerous foreskin cannot clean underneath it — the smegma can be the consequence rather than the cause. Nobody has shown it causes the disease; nobody has shown it is a pure bystander either. “We do not know which way round” is the accurate sentence, and it is still reassuring if you wash, because hygiene is the part you can act on.
What actually drives penile cancer. 95% of infiltrating penile cancers are squamous cell carcinomas, one-third of them related to human papillomavirus (Neuville, 2024). The major, synergistic risk factors are male genital lichen sclerosus and persistent high-risk HPV, and the same review lists phimosis, smoking and poor genital hygiene as additional modifiable risk factors (Uppal, 2026). HPV is the primary risk factor (Stelmach, 2026), and HPV and p16 status carry prognostic weight once disease exists (Vandermaesen, 2025).
Read that third item as written. It is poor genital hygiene — years of a space that is never cleaned, usually with a foreskin too tight to clean it — not a normal daily secretion in a man who washes. And it is listed as modifiable. That word is why I bother writing any of this: the thirty-second habit below is not a nicety, it is the intervention, and it is the one part of that list you hold entirely in your own hands.
Your partner. Cervical cancer is an HPV disease. Male circumcision is associated with lower odds of it in female partners across 19 observational studies (odds ratio 0.65, 95% CI 0.53 to 0.79), and those authors say the effect of circumcision on HPV infection still needs to be understood (Qurieshi, 2025). That analysis is about circumcision status; it did not measure smegma. The penile microbiome is studied as a factor in a partner’s risk of bacterial vaginosis and STIs (Mehta, 2026), and reviews of penile microbiota discuss its impact on the cervicovaginal microbiota (Onywera, 2020). What changes your partner’s risk is HPV vaccination, for both of you.
And the corollary. If smegma is not the risk, something else is. Penile cancer is largely preventable — early treatment of lichen sclerosus and precursor lesions, HPV vaccination, circumcision, smoking cessation — yet it remains under-recognised in older men, with mortality particularly high over 75 (Uppal, 2026). Caught late it is a bad disease: five-year survival in stage IV is 16% (Stelmach, 2026), and lymph node involvement is the main prognostic factor (Neuville, 2024). So a lump, a wart, a white or red patch, or a sore that has not healed in three or four weeks is not a hygiene problem. Do not watch it at home and do not treat it blind. It is the row I most want you to read in the table below.
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How to clean under the foreskin, step by step
Thirty seconds a day. That is the whole plan, and it is the answer to almost everything above. I teach this routine several times a week, and it is the single most useful thing I do in these consultations.
Six steps, and the last one is the one men forget. Pin it somewhere if you have to.
In full, because the details are where people go wrong.
Wash your hands first. You are about to touch mucosa.
Use warm water, not hot. Hot water strips the skin and leaves it irritated.
Retract the foreskin gently — only as far as it moves freely and without pain. Never force it. Forcing tears the delicate inner skin, and that scar is how a normal foreskin becomes a tight one. I see the end result of that mistake often enough that I say this twice in every consultation.
Wash underneath with water, plus a little mild, unscented soap if you want it. An uncircumcised penis requires no special care: normal washing with soap and water, plus gentle retraction during bathing, is what the paediatric urology literature recommends (McGregor, 2007). Plain warm water does the job; a strong soap is worse than none.
Rinse completely. Left-behind soap is a common cause of the itching people then blame on smegma.
Pat completely dry with a clean, soft towel. Moisture trapped in a closed fold is what bacteria and yeast want.
Always return the foreskin forward over the head. Non-negotiable, and the step I most often find has been skipped. A foreskin left retracted behind the glans can form a tight constricting band that causes swelling of the distal penis and acute discomfort (Hohlfeld, 2021) — that is paraphimosis, and it is treated as a medical emergency (Dobanovacki, 2012). I would rather you remembered this one line than anything else on the page.
Circumcised men: wash the exposed head and surrounding skin gently. Everyone: include the shaft, base and scrotum, where those folds trap sweat, often the real source of the smell.
Repeat daily, and after sweating or sex. Anyone caring for an elderly, bed-bound or catheterised man should do the same on his behalf, including returning the foreskin — routinely forgotten.
Hardened smegma, and what to avoid
In a man who has not cleaned the space for a long time, smegma can dry into a firm mass that will not rinse away and restricts the foreskin’s movement. I see this a few times a year, and the men it happens to are usually the most embarrassed people in my waiting room. They should not be.
Soften it, do not attack it. A warm bath or long shower helps. If that is not enough, apply a small amount of a bland, unscented emollient or plain mineral oil — the point is to soften, not to medicate — rinse after a few minutes, and repeat over several days. Two caveats the pages recommending this never give you: it is a softening agent, not a treatment; and mineral and vegetable oils degrade latex, so rinse thoroughly and do not rely on a condom immediately afterwards.
Do not scrub, scratch or scrape. The glans is mucosa, not heel skin. Smegma found on physical examination has been associated with epithelial and stromal inflammation of the foreskin — in a Rakai cohort of men with and without HIV and HSV-2, with the direction of that association unestablished (Johnson, 2009). Either way, abrading an inflamed mucosal surface is the wrong move.
Avoid scented products. Perfumed soaps, deodorants, talc, bubble bath, scented wipes and intimate sprays mask a smell rather than fix it. The evidence here is from vulvar dermatology rather than the male side, which has not been written: cleansing practices, product selection and environmental factors contribute to irritation and are implicated in contact dermatitis (Thakker, 2026). I extend it to men because the skin biology is the same and it matches what I see.
Avoid the home remedies the health sites keep recommending. Tea tree oil, apple cider vinegar soaks, salt-water soaks and neat aloe appear repeatedly on pages ranking for this query. Do not put them on genital mucosa. The distinction from the bland oil above is not arbitrary: an unscented emollient is an occlusive softener, these are irritants on mucosa. What I see is chemical irritation on top of a problem plain water would have fixed. And wear clean, loose cotton underwear.
Smegma in women
Same material, same principle, different folds.
Separate the outer and inner labia gently with clean fingers and wash around the folds and the clitoral hood with warm water and, if you wish, a mild unscented cleanser. Keep soap and water out of the vaginal canal — the outside is what needs washing, not the inside. Pat the outer skin thoroughly dry. Vulvar hygiene and personal care habits play a significant role in vulvar health and in preventing irritant and allergic reactions (Thakker, 2026), so for recurrent irritation the fix is usually fewer products, not more.
If buildup under the clitoral hood is uncomfortable, or the hood has become adherent, have it examined. Thick itchy discharge with soreness is more likely thrush: vulvovaginal candidiasis is one of the most common causes of infectious vaginitis, and approximately three-quarters of women have at least one episode in their lifetime (Ono, 2009). It responds to antifungal treatment (Nyirjesy, 2013).
Boys and babies: do not force the foreskin back
This is the section I would most like every parent in India to read.
A baby’s foreskin is supposed to be non-retractile. I want that sentence read twice, because almost every scarred foreskin I treat in a boy began with an adult not knowing it. At birth it is fused to the glans. Those are physiological preputial adhesions and they separate gradually, on their own, over years. Telling that normal state apart from true pathological phimosis — the kind caused by fibrosis and scarring — is the whole task, because the first is managed conservatively and the second requires surgical management (McGregor, 2007; Dobanovacki, 2012). Among boys referred to a regional paediatric surgery clinic with foreskin symptoms, at a median age of 7.2 years, 77% had physiological phimosis and 18% the pathological kind (Sutton, 2023).
The age anchor, and there is only one in this literature: “up to six years of age the tip of the prepuce is elastic and might be retracted by gentle manipulations” (Dobanovacki, 2012). Think early childhood, not infancy — and note that even at seven or eight, non-retractility on its own is not what needs a referral. A symptom is.
Before age one, wash only the outside, as part of an ordinary bath. Make no attempt to retract.
Never force it back, at any age. Forced retraction causes pain, tears the fusion line, and can leave the foreskin trapped behind the head. In my own clinic the genuinely scarred, tight foreskins I see in boys have almost always been pulled back by an adult first. And once a foreskin is pathologically phimotic it is treated less reliably than parents expect: in a real-world cohort of 214 boys, only about a third responded to first-line topical betamethasone, and of the 18 with clinical signs of lichen sclerosus at presentation, none had a definitive response and 16 needed surgery (Tobia Gonzalez, 2026). That is the cost of pulling it back.
A yellowish-white lump under the skin is almost certainly a smegma pearl — shed cells collecting under an adhesion. Benign, and the treatment is reassurance (Baral, 2023). The same material turns up under residual adhesions and skin bridges after circumcision (Baky Fahmy, 2024).
When a boy’s foreskin does need looking at. Ballooning with a weak or straining urine stream. Repeated redness and soreness. Pain on passing urine. And the one that matters most: a white, thickened, scarred-looking ring at the tip — that is lichen sclerosus, it does not resolve with time, and it is the finding most strongly associated with failed medical treatment and surgery (Tobia Gonzalez, 2026). With none of those present, non-retractility alone is not a reason to be referred: over 75% of boys referred have a normal foreskin, over half are discharged at the first review, and better information for parents and GPs would prevent most of those journeys (Sutton, 2023).
Dr Shah’s notes (from my clinical observation)
Three patterns account for nearly everything that walks into my clinic with this complaint.
The over-washer. A young man decides he is unclean and washes four times a day with a strong antibacterial soap. He arrives with a red, raw, stinging glans, convinced the infection is worsening. It is not an infection. Plain water only for two weeks, and it resolves. Over-cleaning causes more balanitis in my practice than under-cleaning.
The frightened teenager. Fifteen or sixteen, brought by a worried parent, having read that smegma causes cancer, and scraping at it with a fingernail. I show him the anatomy, teach him the routine, and say out loud that he has done nothing wrong. That sentence is the treatment.
The man whose sugars are wrong. Middle-aged, recurrent buildup, foreskin tightening over a year, soreness returning after each tube of cream from the chemist. In my clinic that pattern has repeatedly turned out to be undiagnosed diabetes, and the useful test is a fasting glucose. That is my experience talking, not a published yield figure.
When to see a doctor
Almost all of this needs only a habit. Here is the line.
| What you are seeing | What it means | What to do |
|---|---|---|
| White paste, no smell after washing | Normal secretion | Wash daily |
| Returns in a day or two, rinses off easily | Normal production | Nothing |
| Smell clears within a few days of daily washing | Simple accumulation | Keep washing |
| Marked redness, swelling or soreness | Balanitis or balanoposthitis | Get seen this week |
| Cheesy discharge or odour still there after a week of careful washing | Not simple smegma | Get examined |
| Thick urethral discharge, burning, or a new ulcer | Possible STI — and any ulcer that does not heal needs re-examination whatever the STI result | Get tested now; go back if it has not healed |
| Any lump, wart, white or red patch, or sore not healed in 3–4 weeks — at any age, especially over 50 | Needs a definite diagnosis. This is how precursor lesions and, rarely, penile cancer present, and it is under-recognised in older men (Uppal, 2026) | Get examined in person — not an STI screen alone, and never treated blind |
| In an adult: foreskin will not retract, or splits and bleeds | Pathological phimosis or lichen sclerosus | Needs assessment |
| In a boy: ballooning with a weak stream, repeated soreness, or a white scarred ring at the tip | Not ordinary physiological phimosis | Needs assessment. Non-retractility on its own does not (Sutton, 2023) |
| Foreskin stuck behind the head, swollen, painful | Paraphimosis | Emergency department now |
The examination takes a minute. If the skin is inflamed I establish whether it is a yeast, bacterial, irritant or inflammatory-dermatosis picture and treat accordingly, always alongside finding the cause. If there is a lesion rather than inflammation, the answer is not a cream at all — it is a proper look and, where there is any doubt, a biopsy, because that is what separates a benign inflammatory condition from a premalignant one. Recurrent inflammation is where balanitis shades into something worth naming.
None of that is a reason to inspect yourself anxiously every evening. It is a short list of things worth acting on, so that everything not on it can stop occupying your attention.
Have it looked at once, properly
Redness that keeps returning, a foreskin getting tighter, or any sore that has not healed in a month is worth a single in-person examination.
Frequently asked questions
Is it okay to peel off smegma?
Peel, no. Wash, yes. Using a nail or force to lift material stuck to the glans tears mucosa and scars it — I have seen the scarring that follows, and it lasts far longer than the buildup did. What is loose comes away with warm water and a fingertip; what is stuck should be softened over days.
Does smegma mean poor hygiene?
Not by itself. Everyone with a foreskin produces it daily, including men who shower twice. Heavy accumulation with odour means it has not been washed off recently. Sustained poor hygiene over years is a different thing, and it is a listed modifiable risk factor for penile cancer (Uppal, 2026) — which is why the daily habit is worth having.
Does smegma carry sperm?
No. Smegma is shed epithelial cells from the foreskin and glans; semen is made in the testes and accessory glands and leaves through the urethra. Different material, different place. It cannot cause pregnancy, and I answer this one often enough that it plainly deserves saying out loud.
Can smegma go away permanently?
No — and it should not. As long as you have skin in that fold you will make it. No procedure, cream or diet stops production; you manage it to zero visible buildup with thirty seconds a day. Circumcision removes the fold, but it is an operation with its own indications, not a hygiene treatment.
Is too much smegma harmful?
Not in itself. It becomes a problem only when left to accumulate — that is when it smells, irritates the skin and can set off balanitis.
What does smegma smell like?
Cheesy, slightly sour, sometimes musky or like sour milk. The odour is bacterial rather than the smegma itself: the subpreputial space is normally colonised by a wide range of organisms (Chung, 2019), and it is warm and enclosed. It goes within a few days of proper washing.
Can I remove smegma with my fingers?
Yes — gently, with clean hands, in warm water, using the pad of a fingertip. Never a nail, brush, cloth or cotton bud, and never on skin that is stuck.
Why am I producing so much smegma?
Usually heat and humidity, incomplete washing, or a foreskin too tight to reach the space. Adolescence increases it. A sudden increase with soreness is worth a fasting glucose — in my clinic that pattern turns up undiagnosed diabetes often enough to justify the test.
Will smegma go away by itself?
The accumulation will, once you wash it. The production will not — this is a secretion with a daily output, not a disease with a course.
Does smegma mean I have an STD?
No. It is not sexually transmitted and not contagious. Urethral discharge, burning on passing urine, ulcers or new lumps are different findings and do warrant testing at an STI clinic — and if you are counting days since a specific exposure, the window period decides when a test actually means anything. A sore or lump that has not healed in a month needs examining even if the STI tests come back clear.
Is it okay to have smegma at 14?
Completely. Sebaceous activity rises with puberty, so this is the age most boys first notice it. Wash daily, do not force a foreskin that does not retract yet, and do not scrape. If you are fourteen and reading this worried, I can tell you plainly that nothing is wrong with you — and the other thing boys this age quietly worry about, how often is too often, I have answered separately.
Is it dirtier to be uncircumcised?
No — a different routine, not a lower standard. One anatomy has a fold needing thirty seconds a day; the other does not. The bacterial populations differ (Onywera, 2020), but a washed uncircumcised penis is not unhygienic.
How do I clean under a very tight foreskin?
Retract only as far as it goes without pain and rinse what you can reach under a gentle stream of warm water. Do not force it or use anything pressurised. Then get the tightness assessed: in a child it is usually normal for the age and needs reassurance rather than surgery (McGregor, 2007), while in an adult a foreskin that has become tight is not a stage that will pass, and I would rather see you about it early than after it has scarred.
You have not done anything wrong
I will end where I began, because it is the sentence most men come here needing. Smegma is your body working normally. Not dirt, not an infection, not an STI, and not a cause of cancer that anyone has demonstrated. The treatment is a half-minute habit, and its most important part is the gentlest — never forcing anything, always rolling the foreskin back down.
Two things are worth carrying away. Wash the space daily, because good genital hygiene is on the short list of things that are genuinely modifiable. And if you ever find a sore, a lump or a patch that has not healed in a month, do not wash it, wait on it or put a cream on it — come and have it examined. In my clinic most men leave with nothing more than an explanation, and I would much rather give you that than have you sit at home reading.
References
- Anyanwu LJ, et al. (2012). Microbiology of smegma in boys in Kano, Nigeria. J Surg Res. PMID 21872267
- Baky Fahmy MA, et al. (2024). Spectrum and management of glanular-preputial adhesions after ritual male circumcision. BMC Urol. PMID 39716167
- Baral K, Poudel S. (2023). Smegma pearl: a benign penile lesion in infants. Clin Case Rep. PMID 37361659
- Barry R, et al. (2026). Male genital lichen sclerosus misreported as candidal or other infective balanitides. Int J STD AIDS. PMID 41026603
- Brinton LA, et al. (1991). Risk factors for penile cancer: results from a case-control study in China. Int J Cancer. PMID 1995481
- Chung JM, et al. (2019). Microbiology of smegma: prospective comparative control study. Investig Clin Urol. PMID 30838346
- Demir S, et al. (2020). Microorganisms and antibiotic profile of the subpreputial space in uncircumcised boys. Urol J. PMID 33236338
- Dobanovacki D, et al. (2012). Prepuce in boys and adolescents: what, when, and how? Med Pregl. PMID 22924249
- Hohlfeld A, et al. (2021). Circumcision devices versus standard surgical techniques in adolescent and adult male circumcisions. Cochrane Database Syst Rev. PMID 33786810
- Johnson KE, et al. (2009). Foreskin inflammation is associated with HIV and HSV-2 infections in Rakai, Uganda. AIDS. PMID 19584700
- Leeson C, et al. (2025). Foreskin care: hygiene, importance of counselling, and management of common complications. Can Fam Physician. PMID 39965976
- McGregor TB, et al. (2007). Pathologic and physiologic phimosis: approach to the phimotic foreskin. Can Fam Physician. PMID 17872680
- Mehta SD. (2026). Role of the penile microbiome in female sex partner risk of bacterial vaginosis and STIs. Clin Microbiol Rev. PMID 42233652
- Mizelle DL, et al. (2026). A thematic analysis of heterosexual Black men’s genital hygiene messages. Health Commun. PMID 40391392
- Neuville P, et al. (2024). French AFU Cancer Committee guidelines — update 2024–2026: penile cancer. Fr J Urol. PMID 39581662
- Nyirjesy P, Sobel JD. (2013). Genital mycotic infections in patients with diabetes. Postgrad Med. PMID 23748505
- Ono F, Yasumoto S. (2009). Genital candidiasis. Nihon Rinsho. PMID 19177766
- Onywera H, et al. (2020). The penile microbiota in uncircumcised and circumcised men. Front Med. PMID 32850898
- Qurieshi MA, et al. (2025). Association between male circumcision and cervical cancer risk: systematic review and meta-analysis. JCO Glob Oncol. PMID 41343744
- Stary A, et al. (1996). Oral fluconazole versus topical clotrimazole in patients with candida balanitis. Genitourin Med. PMID 8698375
- Stelmach R, et al. (2026). Management of advanced penile cancer. Oncol Res Treat. PMID 40324348
- Sutton G, et al. (2023). Referrals from primary care with foreskin symptoms: room for improvement. J Pediatr Surg. PMID 36428185
- Thakker S, et al. (2026). Approach to the patient with vulvar disease. Clin Obstet Gynecol. PMID 41662480
- Tobia Gonzalez S, et al. (2026). Real-world outcomes of topical corticosteroids for pediatric phimosis. J Pediatr Urol. PMID 42424714
- Uppal E, et al. (2026). Penile cancer — a preventable cause of death in elderly men. Br J Hosp Med. PMID 41914004
- Vandermaesen K, et al. (2025). The prognostic role of HPV status in penile squamous cell carcinoma. Int J Impot Res. PMID 40770564
A private, judgment-free space to talk through fertility and men’s sexual health. Walk in, or book ahead by phone.
📍No 21, Sree Kalki Apartments, Ground Floor, Bazullah Road, T-Nagar, Chennai 600017